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doi: 10.1038/s41366-022-01085-4

These proinflammatory molecules also induce the above lesions through the nuclear factor-B (NF-B), Janus kinase/signal transducers and activators of transcription (JAK/STAT), and TGF-/Smad signaling pathways[99-101]

Semin Cancer Biol 75:314 Kim I, Shu CW, Xu W, Shiau CW, Grant D, Vasile S et al (2009) Chemical biology investigation of cell death pathways activated by endoplasmic reticulum stress reveals cytoprotective modulators of ASK1

Alcohol Consumption and Prevalence of Human Papillomavirus (HPV) Infection among US Men in the HPV in Men (HIM) Study