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This regulatory mechanism is corroborated by findings in myocardial injury models: miR-182 promotes M2 polarization via the TLR4/NF-B/PI3K signaling axis, thereby improving myocardial ischemiareperfusion injury (133), whereas miR-155 promotes M1 polarization through TLR signaling, exacerbating myocardial inflammation (134), collectively highlighting a conserved role of miRNAs in regulating macrophage polarization

Long-term B12 deficiency can result in more severe complications, such as nerve damage and anemia

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TB500 promotes cell migration, cytoskeletal remodeling, and new vessel growth, aiding tissue elasticity, reducing fibrotic scarring, and supporting endothelial health

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