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Histopathological changes in liver and kidney tissue were alleviated by losartan

aureus (less white) and the GSNO hydrogel also grows less bacteria compared to the hydrogel with buffer or CD

Initially, the pathogenesis of NASH was explained by the 2-hit hypothesis, where the first hit, hepatic triglyceride accumulation (steatosis), increases the livers susceptibility to injury from second hits, such as inflammatory cytokines, mitochondrial dysfunction, and oxidative stress [37,38]

Abstract Lysine methylation facilitates protein-protein interactions through the activity of methyllysine (Kme) reader proteins