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Description
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In addition, this review discusses the multilevel pathophysiological mechanisms involved in acute pancreatitis, including aberrant enzymatic activation, calcium overload, impaired autophagy, inflammatory responses, and various forms of pancreatic acinar cell death
Background Postprandial hypoglycemia (PH) is a significant complication following upper gastrointestinal (GI) surgery, characterized by postprandial glucose fluctuations and diverse clinical manifestations
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