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Use the correct dosage: Start with the lowest possible dose, then slowly increase and only under medical supervision

The observation of widespread RCD in the brain of pre-clinical in vivo developmental models prompted further investigations to find the causative underlying pathways which precede the activation (Figure 6)

The most studied mechanisms include: upregulation of growth hormone receptors in fibroblasts (increasing sensitivity to healing signals), activation of the FAK-paxillin pathway (promoting cell adhesion and migration), modulation of the nitric oxide system through Src-Caveolin-1-eNOS phosphorylation (enhancing vascular function), and dual activation of the Egr-1/NAB2 gene loop (promoting organized, non-chaotic blood vessel growth)

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