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(2011) studied the effect of sleep on L5 apical tuft spine dynamics in adolescent Thy1-GFP mice using a thinned skull approach

3 Pathological Mechanism and Risk Factors of Ferroptosis In fact, among the many pathological mechanisms of delayed wound healing in diabetic ulcers, the idea that hyperglycemia-induced free radicals and oxidative stress lead to cell death and exacerbate the delayed healing of diabetic ulcers has received extensive attention in recent years

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Membranes were probed overnight at 4 C with primary antibodies against p-c-Met (3077 T, Cell signaling), c-Met (8198 S, Cell signaling), p-AKT (AF0016, affinity), AKT (GR100134-1, abcam), p-ERK (AF1015, affinity), ERK (4695 T, Cell signaling), GAPDH (10494-1-AP, proteintech), p-TRKB (4603 T, Cell signaling), CyclinD1 (26939-1-AP, proteintech), cleaved caspase-3 (9661 L, Cell signaling), caspase-3 (66470-2-Ig, proteintech), N-cadherin (22018-1-ap, proteintech), E-cadherin (AF0138, Beyotime), -actin (A5441, Sigma), AXL (4977, CST), p-MER (AF8443, affinity), P-AXL (334580, abmart), NF-KB (10745-1-AP, proteintech), p-ALK (3341 S, CST), Ki67 (27309-1-AP, proteintech)