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glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNFα-mediated cytotoxicity via IKK-β S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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[DOI] [PubMed] [Google Scholar] 50.Sprague JE, Nichols DE

glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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glutathione in liver diseases and hepatotoxicity disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non-alcoholic fatty disease Frontiers | Acetaminophen-induced liver injury:

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