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Nicotinamide Metabolism Mediates Resistance to Venetoclax in Relapsed Acute Myeloid Leukemia Stem Cells

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The activation of CD36 can trigger multiple proinflammatory and profibrotic pathways, such as those involving Toll-like receptors, the NLRP3 inflammasome, PKC-NADPH oxidase, Src family kinases, MAPK, and TGF- [74]