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This concept was supported by the observation that INS-1 cells stimulated with a high glucose concentration exhibited an increased efflux of citrate from mitochondria to the cytoplasm [40]
Introduction Isoprenaline myocardial infarction in rats is known to be a rapid, simple and non-invasive method that produces myocardial damage similar to that seen in acute myocardial infarction in humans [1]
The impaired mitochondrial function and disturbed fatty acid oxidation accounted for the phenotype of skeletal muscle in TauT/ mice (178)
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