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It works through a different mechanism than GHK-Cu: PDRN activates the A2A adenosine receptor, which triggers tissue regeneration, dampens inflammation, and supports the cellular metabolism of dermal cells

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The mitochondrial translocation of phospho-JNK causes the enhanced leakage of electrons from the electron transport chain and formation of superoxide, which reacts with nitric oxide (NO) to form the highly reactive oxidant peroxynitrite in the mitochondria [49]