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Blocking methionine catabolism induces senescence and confers vulnerability to GSK3 inhibition in liver cancer

Since both glucose and fatty acids could contribute to histone acetylation, we reasoned that this could occur either by two distinct substrate-specific mechanisms or a mechanism in which these substrates can feed into a common precursor acetyl-CoA pool (e.g., in mitochondria) that is then transported to the nuclear-cytosolic compartment (Supplemental Figure 5E)

Allergy 77:16231625 [DOI] [PMC free article] [PubMed] [Google Scholar] 112.Samin A, Al Kury LT, Imran-Khan M, Hussain S, Alattar A, Alshaman R, Shah FA, Aamir M, Li S (2021) Acetyl-l-carnitine protects against LPS induced depression via PPAR- induced inhibition of NF-B/NLRP3 pathway

Tracer amino acids for metabolic and structural studies

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