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Description
Collectively, these clinical cases establish SLC39A8 as an essential Mn transporter and show that mutations can disrupt Mn homeostasis, leading to Mn deficiency and thereby impairing key Mn-dependent processes in glycosylation and mitochondrial antioxidant defense systems
Neurosci 40, 90669077
a National Heart, Lung, and Blood Institute (NHLBI) grant no
Hu et al 2016
Adane and colleagues reported the presence of NOX2, a ROS-generating enzyme that is localized to the cytoplasm, in both bulk AML cells and LSCs [82]
