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Description
The latest research indicates that I3A not only sustains lung epithelial barrier function through AhR receptor activation and IL-22 secretion stimulation but also inhibits HDAC5/6, resulting in the inactivation of the NF-B/NLRP3 signaling pathway and the subsequent reduction of inflammatory factor expression ( 3.4 Other pneumonias: unique pathogenic mechanisms of mycoplasma, chlamydia, and mixed infections For atypical pathogens, such as Mycoplasma pneumoniae , or in cases of mixed infections, the regulatory mechanisms of the intestinal flora exhibit significant diversity

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