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doi:10.1016/j.sjbs.2020.01.012 Wang F, Wang S, Zhang Z, et al
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204 Similarly, administration of the PRMT5 inhibitor PJ-68 in a CML mouse model with high expression of PRMT5 LSCs could deplete DVL3, thereby inhibiting Wnt/-catenin signaling and significantly prolonging the survival time of a retroviral BCR-ABL-driven CML mouse model
cells, nanomedicine, Biotechnol
