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The EBV latency III induces resistance to ferroptosis inducing agents To gain insights into how EBV latency programs alter lipid ROS levels and ferroptosis vulnerability, we utilized isogenic Burkitt tumor derived MUTU I vs III cell lines, which differ only by EBV latency I vs III programs, (43)

The pots were kept in submerged conditions by maintaining a water level of 23 cm above the soil surface to create fully anaerobic conditions within the pots

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The discovery that melatonin enhances the survival and function of insulin-producing -cells [136] adds more support to the idea that melatonin plays a role in regulating insulin signaling

Human neutrophils stimulated with exogenous lactate showed increased formation of the NETs