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Given that these species can accumulate not only through increased production but also through decreased clearance, an inadequate antioxidant response, particularly associated with a dysregulation of NRF2 transcriptional activity, could explain their gradual accumulation

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Silencing circ-Snhg11 reduced the therapeutic benefits of Exos, while its overexpression facilitated angiogenesis and inhibited GPX4-driven ferroptosis