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In this work, we found that modulation of the cytoplasmic pH (pH i ) of Escherichia coli provides a thus far unexplored strategy to support resistance

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This discordant signal may be related to the fact that kidney transplant recipients often have concomitant hypertension and activation of the renin-angiotensin system that may have already contributed to significant adverse cardiac remodeling prior to kidney transplantmaking it less likely to observe differences following kidney transplantation (75, 78)

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