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C32 and C88 in PfGrx) could not only form an intramolecular disulfide during catalysis or affect the reactivity of the active-site thiolate but also function as a resolving cysteine for trapped Grx(SSR) species (Fig

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[DOI] [Google Scholar] 361.Maoka T., Kawashima Y., Takaki M

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8.2 Ferroptosis exacerbates central inflammation Neuroinflammation is primarily initiated by astrocytes and microglia in response to factors such as injury, infection, exposure to toxins, or autoimmune reactions

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