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doi: 10.1016/j.ajpath.2013.06.008

James SJ, Cutler P, Melnyk S, Jernigan S, Janak L, Gaylor DW, Neubrander JA (2004) Metabolic biomarkers of increased oxidative stress and impaired methylation capacity in children with autism

NLRP3 activation further appears to be inhibited by the activity of the ketone body HB produced by the KD, suggesting that the modulation of neuroinflammation contributes to KD efficacy (Youm et al., 2015)

Direct comparison of ML226 treatment to lentiviral ABHD11 sgRNA depletion demonstrated that both pharmacological and genetic inhibition of ABHD11 reduced functional lipoylation on the E2 subunit of the OGDHc (dihydrolipoamide succinyltransferase (DLST), referred to as the OGDHc-E2) in preference to the E2 subunit of PDHc (dihydrolipoamide acetyltransferase (DLAT), referred to as the PDHc-E2