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Although preclinical evidence supports the promise of NAD boosters, human clinical trials have shown limited efficacy [215]
However, targeting Nrf2 and its related targets remains a viable approach to prevent or treat DCM by regulating ferroptosis
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vaginalis proteasome as a viable drug target for therapeutic strategies against trichomoniasis and led to a whole new class of compounds to be explored: proteasome inhibitors
Opposing effects of polysulfides and thioredoxin on apoptosis through caspase persulfidation
