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by inducing ENS neuronal inhibition, despite the reported local GABA depolarizing actions
Cell swelling, heat, and chemical agonists use distinct pathways for the activation of the cation channel TRPV4
This antiapoptotic mechanism involves a combination of rapid actions that are independent of nuclear events and that potentially lead to alteration in protein phosphorylation (306, 307) and a more classical genomic mechanism inducing an anti-inflammatory cascade via expression of the orphan receptor, liver receptor homolog-1 (308)
10.4049/jimmunol.177.1.422 42 PereraD
sc-13156), PPAR (0.20 g/ml
