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glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

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Transgenic mouse lines The Aldh1l1-EGFP-Rpl10a transgenic mouse line expressing an EGFP-tagged ribosomal protein l10a under the astrocyte-specific Aldh1l1 promoter was generated as previously described (Doyle et al., 2008) and re-derived on a C57B/6 background from frozen sperm imported from Jackson Laboratories (Mouse Strain Number030248)

glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

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glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

Abudoureyimu M, Sun N, Chen W, Lin X, Pan F, Wang R

glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

PMID: 71 PellissierL.Pinto-FigueroaE.Niculita-HirzelH.MooraM.VillardL.GoudetJ.et al

glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

Dorion S, Lambert H and Landry J

glutathione's role in cataracts Deficiency glutathione peroxidase 4 (GPX4) results in abnormal lens development and newborn cataract Protein kinase A activation alleviates

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