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[DOI] [PubMed] [Google Scholar] 109.Wallace T.C., Bailey R.L., Blumberg J.B., Burton-Freeman B., Chen C.O., Crowe-White K.M., Drewnowski A., Hooshmand S., Johnson E., Lewis R

Therefore, UBB+1 is involved in both secretase-dependent and independent pathways for A generation and this could be a reasonable explanation as to why UBB+ conjugated protein aggregation has been found to be associated with A plaques in transgenic UBB+1 AD brain (van Leeuwen et al., 2006

10.1016/j.cbpc.2010.10.004 45 LushchakV
Rachelefsky, G

Peptides preserve what exists, don't resurrect dead tissue
