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Currently, the changes in CoQ 10 levels in AD patients, and their effects on improving neuropsychological assessments, are not clearly confirmed, and the exact role of CoQ 10 in the pathogenesis of AD and the treatment of neurodegenerative diseases is yet to be clarified [91,128]

2b) and the ChIP-Seq experiment differ, presumably because (1) these methods examine global vs

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Analysis of genes associated with proinflammatory and profibrotic pathways upregulated in ischemia-induced chronic kidney disease in cats
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[PubMed: 2944781] 24
