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17-Estradiol treatment is unable to reproduce p85 redistribution associated with gestational insulin resistance in rats

ME/CFS patient serum induces metabolic changes in myoblasts, including excessive lactate, which indicates that PDH dysfunction underlies PEM and inadequate ATP generation [76]

These changes are accompanied by increases in the levels of salicylic acid (SA), pathogenesis-related proteins, and camalexin that demonstrate the activation of the ROS signalling branch of plant innate immunity (Pavet et al

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