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Studies conducted in vitro and in animals have indicated that -tocopherol and its major metabolite, -carboxyethyl hydroxychroman (-CEHC), may play a role in protecting the body from free radical-induced damage in various conditions of oxidative stress and inflammation (reviewed in 8)

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Experimental work in isolated vascular tissue and cell culture models indicates interactions involving regulators such as endothelial nitric oxide synthase, Src kinase signaling, and vascular endothelial growth factor receptor pathways

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