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Keywords: post-acute sequelae of SARS-CoV-2, COVID-19, Long COVID, inflammation, metabolism, immunosenescence INTRODUCTION Long COVID (LC) is a multisystem condition following the acute phase of COVID-19, in which individuals experience persistent or new COVID-attributed symptoms including fatigue, cognitive impairment and post-exertional malaise (1, 2)

Since the human genome contains only one glutathione synthetase gene, the various clinical forms of glutathione synthetase deficiency reflect different mutations or epigenetic modifications in the glutathione synthetase gene

In addition, this review discusses the multilevel pathophysiological mechanisms involved in acute pancreatitis, including aberrant enzymatic activation, calcium overload, impaired autophagy, inflammatory responses, and various forms of pancreatic acinar cell death