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A pilot study of oral therapy with a 5-LOX inhibitor (zileuton) for AD demonstrated that the compound prevented disease exacerbations and prolonged remission, confirming the functional role of LTB 4 in the development of this disease [229]

In a separate study using mouse and human systems, an increase in inflammatory CD4 + effector T-cell migration into the intestinal mucosa was mediated by D5 heteromerization with the chemokine receptor CCR9

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NAC is assumed to act as a disulfide bond reducer, scavenger of reactive oxygen species, and precursor of glutathione biosynthesis