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Molecular profiling of the residual disease of triple-negative breast cancers after neoadjuvant chemotherapy identifies actionable therapeutic targets

Costa TM, Hermann KL, Garcia-Roman M, Valle RDCSC, Tavares LBB

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U73122 did not affect the increase of phosphorylation of JNK by -toxin and PGN, whereas it completely inhibited the upregulation of G-CSF mRNA expression by -toxin and PGN (Fig

(And even if they were approved, pharmaceutical companies would shy away from them for fear of being undercut by compounding pharmacies.) If companies can sell them and make lots of money without having to prove that they do what theyre claimed to do, why wouldnt they? Zettler points out