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Interestingly, Chac1 expression is decreased in livers of patients with MASLD, highlighting its potential therapeutic relevance

Burden of Disease Collaborators

In addition, some drugs require metabolism to generate an active metabolite or functional therapeutic agent (see Table 1)

In the failing heart, increased levels of GPCR kinases (GRKs) phosphorylate, and thereby desensitize and downregulate, -ARs, resulting in a loss of cardiomyocyte contractile reserve

These senescent cells release harmful pro-inflammatory signals, known as the senescence-associated secretory phenotype (SASP), that damage surrounding tissues and disrupt overall organ function