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Given that YAP1 and TAZ are homologues with overlapping functions as downstream effectors of the Hippo pathway [4], we also observed that co-knockdown of CDK4/6, treatment with Abemaciclib or DUB3 depletion could reduce TAZ protein levels, likely due to increased TAZ polyubiquitination (Figs

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We hypothesize that integrating these agents with innate immune modulators could enhance durability, as innate pathways may perpetuate low-level inflammation even after adaptive immunity is suppressed

Then, 10 l aliquots of these reactions were directly added to 90 l ultrafiltered flavan-3,3,4-triol ( 4 ) to initiate the second step of the two-step in vitro assays with 3,4- cis -leucocyanidin ( 6 ) as described below

Antioxidant treatment has become an essential strategy for the adjuvant treatment of preterm newborns with BPD despite the lack of standardized clinical signs