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Description
Splenic infarction due to transient antiphospholipid antibodies induced by acute epstein-barr virus infection

It also enhances blood flow to damaged tissues, which accelerates recovery

10.3390/nu9090980 163

Conversely, some studies have shown that accumulated mutant p53 can interact with NRF2 to repress SLC7A11 expression, weakening GSH synthesis and rendering these cells more susceptible to oxidative damage (82)

Am J Hematol 93(9):1136-1141, 2018
