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Endoplasmic reticulum stress (ER-stress) by 2-deoxy-D-glucose (2DG) reduces cyclooxygenase-2 (COX-2) expression and N-glycosylation and induces a loss of COX-2 activity via a Src kinase-dependent pathway in rabbit articular chondrocytes

GSH serves as a cofactor for the selenoenzyme GPX4, and inhibiting the de novo synthesis of GSH synthesis induces ferroptosis by inactivating GPX4 [56]

Altered mitochondrial fusion drives defensive glutathione synthesis in cells able to switch to glycolytic ATP production

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