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The inhibition of autophagy via Atg7 deletion, Atg5 knockdown, or pharmacological blockade precipitates a shift toward a pro-inflammatory phenotype, characterized by ameboid morphology and the overproduction of cytokines such as IL-1, IL-6, and TNF [103,104]

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This may explain why less research on the gut microbiome has been conducted in HD than in other NDDs, although recent studies point to the role of gut dysbiosis in disease progression and support the exploration of microbiome-based interventions (172)

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however, currently the levels of products of oxidation such as oxidized lipids, proteins, amino acids and DNA are measured as they are more stable