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Notably, Cag pathogenicity island (PAI)-encoded virulence factor CagA manipulates signaling pathways such as ERK/MAPK, NF-KB, leading to pathologic alterations in cell morphology, adhesion, polarity, proliferation, and motility (Wang et al., 2023)
As mentioned above, the regulation of A by UA may occur secondary to autophagy or microglial activation [217]
