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Induction of ferroptosis in human nasopharyngeal cancer cells by cucurbitacin B: molecular mechanism and therapeutic potential

QMP enhances cellular recycling and plasma half-life despite the absence of L585 To study the importance of L585 in a cellular context, we performed a human endothelial cell-based recycling assay (HERA) 48 using human microvascular endothelial cells (HMEC-1) that overexpress human FcRn (HMEC-1-FcRn) (Fig

Ferroptosis represents a novel form of programmed cell death, distinguished by its reliance on iron-mediated lipid peroxidation as a pivotal mechanism leading to cell death [30]

Peptides may accelerate tissue healing

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