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*Correspondence: Hua-you Luo, [email protected] Zhu, [email protected] Xu, [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Mitochondrial dysfunction in keratinocytes may lead to excessive production of H 2 O 2 and the release of cytochrome c, potentially intensifying the inflammatory response and contributing to structural damage of the epidermis

Skin texture and tone also noticeably improve during this noninvasive procedure

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Astrocyte plasticity revealed by adaptations to severe proteotoxic stress