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A base substitution at the splice acceptor site of intron 5 of the COL1A2 gene activates a cryptic splice site within exon 6 and generates abnormal type I procollagen in a patient with EhlersDanlos syndrome type VII

82 They also observed that in primary BM samples from MM patients, isatuximab significantly depleted tumor plasma cells, CD38 hi Blymphocyte precursors, basophils, and NK lymphocytes

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