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J Neurochem 2008

Myeloid-specific GPX4-haploinsufficient mice develop a lupus-like disease spontaneously, but complete GPX4 ablation in neutrophils results in severe neutropenia without inducing a lupus-like disease

Current knowledge is largely derived from preclinical studies investigating the biological processes involved, with a lack of clinical trials confirming these effects in human subjects

Furthermore, HIF-1 promotes glycolysis while inhibiting mitochondrial oxidative phosphorylation, indirectly destabilizing the electron transport chain and further enhancing ROS accumulation (Liang et al., 2021), thereby further accelerating ferroptosis progression