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Structure [edit] The glutathione binding site, or "G-site", is located in the thioredoxin-like domain of both cytosolic and mitochondrial GSTs

GSK3B inhibition can induce the cysteine/glutamate transporter (xCT), through Phosphoinositide-3 kinases (PI3Ks), which imports cysteine and exports glutamate (an excitatory neurotransmitter), leading to an increase in cerebellar glutamate, which may play a role in epilepsy

In addition to regulating the expression of pro-inflammatory factors, MOTS-c can also exert anti-inflammatory effects by targeting immune cells (such as T cells and macrophages) [8, 61]

4 Challenges and prospects of radiation therapy protection 4.1 Exploration of new targets and improvement of radiation protection capability Targeted toll-like receptors (TLRs): TLRs are expressed in various mammalian cells and can activate and upregulate the NF-B signaling pathway, thereby enhancing cellular radiation resistance