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In the skin, GPX4 deficiency in basal keratinocytes alone can trigger psoriasiform lesions [12]
Considering the longer sequences of noncoding RNA, the impact of 8oxoG modifications may be generally less significant
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Although most of NAPQI is formed in the liver, the kidney also metabolizes APAP to the toxic metabolite and releases cysteine conjugate of APAP into the bile and blood for further elimination in urine [9]
