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[54] in Italy, showed profound impairment of APAP metabolism in children with ASD and was published in 1999, several years prior to the study of Schultz et al
NIH National Library of Medicine National Center for Biotechnology Information
-Bo qun ni kh ro, trnh nh nng trc tip (*) Sn phm khng c tc dng thay th thuc cha bnh
273 During the process of aging, unrepaired mtDNA mutations accumulate, eventually leading to mitochondrial dysfunction and diseases
This work was supported by the Clinical Research Priority Program of the University of Zurich for the CRPP Synapse & Trauma
