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The cytotoxicity of As 2 O 3 occurred through the generation of reactive oxygen species (ROS), thus inhibiting radical scavenging systems would enhance the therapeutic efficacy of As 2 O 3 provided that normal cells were relatively resistant to such a measure

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In contrast, no BRD4 and ASXL1 co-occupancy was observed on H3K4me1-rich enhancers or H3K27me3-rich transcriptionally silenced regions (Fig

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Increased cellular free cholesterol in macrophage-specific Abca1 knock-out mice enhances pro-inflammatory response of macrophages

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GSH can be deployed easily, to help protect military personnel and civilians from wartime and terrorist dispersions of poison gas, radioactive materials, and lethal microbes

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& Weiss, J

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