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These observations suggest that part of the survival propensity of C38 cells may be due to an enhanced capacity to continue to prevent lipid hydroperoxides formation (Fig

doi: 10.3389/fnins.2023.1136143 46 LiFJLongHZZhouZWLuoHYXuSGGaoLC

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These mechanisms include, among others, DNA methylation, small RNAs, and histone modification

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