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doi: 10.1681/ASN.2016030354 189

The foundational study behind this claim Khavinson, Bondarev, and Butyugov (2003), published in the Bulletin of Experimental Biology and Medicine found that Epitalon treatment increased expression of the telomerase catalytic subunit and telomerase enzymatic activity in human fetal fibroblast cultures, with corresponding telomere elongation in cells that had been telomerase-negative

showed that the surface modification of sulfinyl and carbonyl groups caused the generation of another discrete energy level, caused by the increasing degree of surface oxidation

Glutathione as a scavenger of free hemin