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It would also reduce microclot formation due to endothelial damage caused by the positive regulation of NOX2 by EBNA-1 in EBV-transformed endothelial cells or by the activation of TLR3 by EBERs [256, 302, 316, 317]

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GH and IGF-1 are also known to increase the expression of bone morphogenetic proteins 2 and 4 (BMP2, and BMP4) [88, 89], which are involved in mesoderm induction, skeletal patterning, and limb morphogenesis [9092]

Currently, all three agents are approved for use in both first-line and subsequent treatment settings for patients with metastatic ER+/HER2 breast cancer [18]