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Proinsulin translation in pancreas cells requires the activity of the Fe-S cluster enzyme, CDKAL1
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In parallel, non-radical ROS, functioning as more stable, diffusible signaling molecules, can be therapeutically harnessed to combat infection and promote regeneration or, conversely, subverted to foster pathological processes like chronic inflammation, tumor progression, and age-related redox dyshomeostasis under abnormal flux regulation
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