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514 Specifically, oxidative modifications at several cysteine residues within p38, including Cys39, Cys119, Cys162, and Cys211, occur following exposure to oxidative stressors such as H 2 O 2 or the ROS inducer prostaglandin J2

What makes it special is that it is designed to disrupt the interaction between FOXO4 and the tumor-suppressor protein p53 within senescent cells, thereby triggering programmed cell death (apoptosis) of those cells

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the first joins the amino acids, glutamate and glycine to make gamma-glutamylcysteine (GGC) and the second adds a glycine to make glutathione