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Inhibition of casp-1 with VX765 reduces the severity of inflammation and behavioral deficits in the EAE model [329]

This helps to improve the therapeutic efficacy and safety of drugs, reduce the risk of adverse reactions and treatment failure caused by differences in drug quality

By preventing nicotinamide methylation, the compound increases intracellular NAD+ availability, which in turn activates sirtuins and other NAD+-dependent enzymes critical for metabolic homeostasis
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Key Benefits of BPC-157 Supports tissue repair studied for its potential role in healing muscles, tendons, and ligaments
