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The recent identification of new redox-active proteins in humans and yeast that mechanistically parallel the more established redox-active enzymes indicates that there might be further uncharacterized redox pathways throughout the cell
doi: 10.3389/fphar.2019.01597
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In contrast, existing single-target therapies often fail to regulate both ROS and NETs simultaneously, limiting the comprehensiveness and durability of therapeutic outcomes [86]
